Scientists say the coronobius will soon be a common cold

Developing an effective vaccine or drug against a new type of coronary is complicated just as the virus is constantly changing. In about half a year from the outbreak of pandemic, scientists worldwide have recorded 100 different mutations in SARS CoV-2.

Such variations or changes are normal, because the genetic structure of the virus changes and new sub-species appear. Virus characteristics can also change. The consequences can be a weakening of the original virus, but it can also make it more aggressive.

Various varieties also confirm that a pathogen in a certain region of the world causes waves of infection with different severity, and why people react differently to infection.

China, the country from which the virus developed, actually managed to bring the coronary under control with crucial limits of movement. In recent weeks, only people have been tested who have returned from outside.

New infections have recently been reported back in several Chinese provinces, and this time it is clear that they originated in China.

At the same time, the first tests showed that a new version of pathogen is flowing in China. The SARS-CoV-2 virus found in Beijing is slightly different from the one that hit China earlier, Chinese Health Service epidemiologist Zheng Guang told Global Times.

The results now must be compared to tests from other countries so that they can follow the pathogenic origin line.

The virus's current trace took health services literally to the board, on which the imported salmon was filled, to the Sinfadi market in Beijing. But it is unknown where salmon come from, considering that China imports this type of fish from Norway, Chile, Australia, Canada, and the Faroe Islands.

But for the virus to spread to people by salmon, it is quite surprising. So far, it has been determined that the risk of transmission is greater when it comes to mammals such as horses, sheep, rabbits, or cats. When it comes to reptiles, fish, or birds, the risk of transmission is, according to previous studies, too small.

To breed, viruses use waiting cells. When viruses attack, they release genetic information from the nucleus into an infected cell. Thus, body cells reproduce millions of copies of the virus. But with these reproductions, cheating mistakes occur, and any error also changes the genetic code of the virus it mutates.

In principle, the human body is only in a situation to protect itself against such viruses. It produces antibodies, which protect it from viruses and make it immune to disease - causing pathogens. But if pathogen mutates, and antibodies are programmed into the old version of pathogen, then antibodies are less effective.

For the same reason, people regularly get colds, as our bodies have already created antibodies from the previous infection, but we don't have new antibodies for the new pathogen mutation.